I hope it was, too - but if you still have it after clearing your ears, how likely is that? Look at your first post here: you stopped drinking, and then you got tinnitus. Occam's razor applies here, let's talk a little bit about how tinnitus, and alcohol work.
Tinnitus is, probably, at least in most cases, a result of nervous system hyperactivity. The system works by a series of checks and balances, where stimulating chemicals (called
excitatory transmitters) are balanced out by calming chemicals (
inhibitory transmitters). The basic inhibitor is called GABA. Nerve cells have little locks on their outer membrane called
receptors. When GABA is present in its receptor, it "inhibits" the cell: it doesn't pass information along to other nerve cells as quickly. When this mechanism is not working normally, one effect is that cells fire "too quickly", "too much" information is being passed along to other cells. I'm using quotes because "too much" is a judgement: the mental and physical reaction to this extra excitation may be anxiety, nervousness, and neurological manifestations like muscle twitching and tinnitus. But, the body is just doing what it's supposed to be doing; its environment has changed, and it is trying to be
homeostatic, which means "maintain a constant state".
Alcohol is a fairly complex drug, all things considered, but at a really basic level one thing it does is cause an increase in the way GABA functions, which in turn causes a decrease in excitatory activity in the nervous system. This is why booze makes you feel relaxed.
When you consume any substance which acts on GABA in this way for a period of time, homeostasis bites you in the butt: your brain says, "a ha! I'm using GABA much more efficiently than I should be, so I need to decrease it". One mechanism by which this happens is
downregulation. Some of the receptor sites on the nerve cells are "turned off", so that GABA cannot bind to them.
When you are in a state of downregulation and then you suddenly rip away the thing which is causing the increase in GABA function, all hell can break loose: it has a similar effect to taking a stimulating substance.
The good news is that, in general, this process reverses over a period of time; homeostasis again kicks in and tries to normalize things. But, tinnitus is a very complex situation: the sound is a result of excessive synchronous firing of nerve cells. This may originate in the cochlea, or the auditory cortex, but very rapidly, the whole brain is involved. One problem is that the brain has a tendency to reinforce pathways which it decides are "important", there is an old saying, "if it fires, it wires" -- meaning, if you do something which causes your brain to fire in a certain way, that firing can get "wired in". One way that this happens is called
long-term potentiation, which is thought to be heavily involved in the maintenance of tinnitus (
https://en.wikipedia.org/wiki/Long-term_potentiation).
So, my question is, how much were you drinking, on average? How many days a week did you drink? How long had you been drinking in that way?
In general, from the anecdotes I have read, tinnitus as a result of alcohol cessation tends to resolve more quickly than tinnitus which arises from drugs like Valium, but there are no hard and fast rules.
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