Found it. It's a continuation in greater detail of the 1996 case of a woman that was treated in Brazil. I don't remember where I found it and I find it very odd that I can't find it online now, but its most probably that its the real article itself I find it highly unlikely that it was tampered with. There is also talk in the literature of a paper in 1998 that was 5 pages long and it was published somewhere but it's not easily findable online, so it's very probable that this is it. Also it is a full objective tinnitus because it was heard from the outside by other people. This is what it says:
''Because our patient complained of continuous, highfrequency tinnitus since childhood, and because we had not seen or read about a high-frequency and continuous sound originating from a muscle and lasting for such a long period, we originally did not consider a diagnosis of middle ear myoclonus. However, informal conversations with bioengineers made us aware that these middle ear muscles might have been contracting in a very specific way, making brief and rapid contractions with short intervals between them, simulating an actual continuous sound.''
So I'm ambivalent about this, because even though it's continuous, it was heard by the doctor and her buddies since she was 14 years old. So it doesn't really help me because mine isn't fully objective. And from asking around it would seem my reflex predominant sound intolerance might be closer to 'hyperacusis' and Silverstein's operation would be closer to home. So on the one hand maybe its the stapes thats out of whack and that's 'hyperacusis' and a matter for Silverstein, and on the other, there are plenty of MEM cases that aren't heard by an external person so we can just combine those cases with this one. However it does fulfill that requirement of making a mockery of the bullshit difference between pulsatile and the sound therapy infested ear-nihilistic 'neuro' tinnitus that has the whooshers crowd
trying to pull an inverse Rosa Parks on our asses.
The paper goes on to talk about a couple of things that might be of interest for those of us with low frequency continuous vibration.
''Earlier reports involving this kind of tinnitus suggested a vascular origin, despite the absence of pulsatility. This mechanism was explained by Glanville et al in 1971.12 According to the authors, the vessel's fibers may remain in a prolonged state of stress in a specific region of continuous flow, generating a nonpulsatile tonal sound. This would occur despite the high elasticity of the vessel's walls because such elasticity would be limited by adjacent structures, causing a state of continual stress in the fibers.''
Both of which make a mockery of ENT and sound therapy practices everywhere, the idea MEM can be continuous, or that tinnitus can be somatosensory, just because this lot looked into it while millions from U.S. army funds are invested into sound therapy from 'experts' that don't know this doesn't really paint an encouraging picture of medicine at large.
There may also be others I'm not sure. The papers on middle ear myoclonus that add a measly one or two cases are so thin they often include summaries of all previous cases, some talk of things like buzzing and static that may or may not also be continuous.
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