Been investigating more about the effect of alcohol on neurotranmission because a therpaist told me that 'alcohol is the antidote to glutamate'. Seems she's right as its a Glutamate antagonist. I'm looking into it as alcohol seems to make my T worse so this could mean that my particular mechanism isnt over-production of glutamate leading to excitotoxicity (which is one of the theories as to the cause of T). Having said that, alcohol does give a temporary threshold shift to the hearing and so this might be why my T gets worse. Thinking about it, when I have a drink the noise I get is different to the normal T. Still no wiser. Anyway, this is the link, and the interesting bit quoted.......
http://pubs.niaaa.nih.gov/publications/arh21-2/120.pdf
"Consistent with the finding that alcohol inhibits
NMDA receptor function, acute alcohol exposure was
shown to reduce excitotoxicity in neurons from the brain's
outer layer (i.e., the cortex) (Tsai et al. 1995). To compensate
for chronic alcohol-induced NMDA receptor inhibition,
however, the number of NMDA receptors on the cells and,
thus, the level of receptor activity increase after long-term
alcohol exposure (Grant et al. 1990; Trujillo and Akil 1995)."