Help Needed for Tinnitus Guide Project

Like Eileen Fehily, I have 30+ years of experience in business communications. I now work as a freelance copy editor. If Eileen (or whomever) needs a backup at times, I can help with editing. I've had tinnitus since 2010.
 
How do we define "evidence-based"?

I doubt we'll make any progress on tinnitus with evidence-based findings. Basically, almost nothing meets the requirements of the Cochrane standards.
You don't even know if corticoids are effective for sudden unilateral hearing loss:
https://www.ncbi.nlm.nih.gov/pubmed/16437471
Not to mention sudden unilateral tinnitus... ;)
 
Yeah. I get tired of the old there is no research to back this or that...
 
[USERGROUP=4]@Moderators[/USERGROUP],

I seriously want to help on this, but in all honestly I'm a bit confused about the format but will work with it.

I can add useful evidence based information to the following sections.

2. What is Tinnitus

3. Hyperacusis and Ear Pain

7. Tinnitus Models

10. Tinnitus Cure

11. Do your research

17. Protecting Your Ears

Also I'd appreciate if we added a section on hearing loss since that is a major role in tinnitus.

Please have someone review my post to make sure they are factual.

Thank you all for allowing me to help.
 
Tinnitus is a malfunction of the auditory brain primarily caused by hearing loss but also TMD, cervical issues and traumatic brain injury perhaps all can be intermixed. Lack of GABA seems to play a role in the development of hearing loss induced tinnitus. A hypothesis suggest a non-painful variation of hyperacusis called "Recruitment" may be the same mechanism as tinnitus in which the brain is acting as an internal hearing aid to compensate for hearing loss. If so tinnitus/recruitment would be the same mechanism.

Painful hyperacusis is a different condition all together linked to peripheral damage where as this is neurological.




https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4208401/
https://www.sciencedirect.com/science/article/pii/S0149763410001843

I believe this combined with GABA hypothesis explains some mechanisms of tinnitus and possibly "non-painful hyperacusis (recruitment)

Any form of hearing loss equals less input given to the dorsal cochlear nucleus which causes the dorsal cochlear nucleus to display hyperactive fusiform cell activity (tinnitus)

http://www.jneurosci.org/content/22/6/2383.long
Proof of this in animal models

A hypothesis suggest without GABA to suppress it it becomes chronic without a treatment.



https://www.tinnitustalk.com/threads/gaba-supplements-including-picamilon.371/
https://www.tinnitustalk.com/threads/clonazepam-klonopin-rivotril.423/

The BTA's website explains how TMD effects hearing and auditory parts if the brain.
https://www.tinnitus.org.uk/tinnitus-and-tmj

Cervical issues can modulate the auditory brain.
https://www.dizziness-and-balance.com/disorders/hearing/tinnitus/cervical tinnitus.html

And head trauma (TBI traumautic brain injury) also plays a role by directly damaging hearing regions of the brain.


I am also going to go as far as saying curing hearing loss or increasing GABA would reduce tinnitus by the hyper fusiform cell activity actually lowering.

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Read here for why I believe the evidence suggest that curing hearing loss is the most effective answer for many patients
https://www.tinnitustalk.com/posts/360596/

Please consider adding this information.
 
I agree with your first video 'What causes tinnitus'. For most, I would stretch the neck for occipital nerve release. Pull the SCM muscles and use good posture.
I am also going to go as far as saying curing hearing loss or increasing GABA would reduce tinnitus
by the hyper fusiform cell activity actually lowering.
I also agree with you on GABA. It has been proven by a company that was given fast track status for a clinical 3 trial. Approval by year's end for a depression drug with GABA influence. They also have other trials and studies.
 

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