New University of Michigan Tinnitus Discovery — Signal Timing

Sorry I need to ask again. Does the device of Susan Shores Team help only for people who can lessen by special movements? If I do some movements (jaw clenching etc.), my tinnitus increases.
 
Sorry I need to ask again. Does the device of Susan Shores Team help only for people who can lessen by special movements? If I do some movements (jaw clenching etc.), my tinnitus increases.

No the device helps anyone who can change their tinnitus via movement. So applies to either increases for decreases or changes to the pitch.
 
Here's a cool video I found explaining the science behind tinnitus and the device



@kelpiemsp did you have a session two days ago? How did it go?

Also, @kelpiemsp this previous comment was intended at the Minnesota thread and I accidently mixed them up because they both have "University of Mi..." in their titles. Sorry about the confusion.
 
Somehow I have a feeling that signal timing could alleviate / remove the symptoms of tinnitus...

But that Frequency TX is working on the cure: regrowing hair cells.

Given the choice, I'd give Frequency TX a first shot. Who knows, perhaps signal timing turns off more than we want in the brain... eliminating the chance of restoring hearing in that frequency.

Just a thought :) I'm not a scientist.
Does signal timing affect hearing in anyway?
 
Does signal timing affect hearing in anyway?
Good question!

If these overactive fusiform cells is what is putting our hearing into overdrive due to the lack of cochlear input and makes us hear more than there is sound in the environment, then I would say yes, signal timing does affect hearing. This is an obvious conclusion you can make if you subscribe to this theory.

I do believe that somato-auditory stimulation will help alleviate tinnitus. But I worry that one may have to use it all the time unless the cochlear input is restored. So at least in people with hearing loss, I think that restoring cochlear input by repairing the hair cells and neural cells should be the first step. If you achieve good results with that, you may not even need the somato-auditory stimulation at all. Your tinnitus may fade away all on its own.

So we now have two groups of scientists that are trying to address the problem from two different angles. One is trying to restore auditory input at the cochlea, and another is going in opposite direction and trying to calm down overactive neurons. Perhaps the solution is somewhere in-between the two or double folded? This makes me feel optimistic about the future.

The only thing missing that I would like to see more development on is:
1. Objective diagnosis of hearing loss
2. Objective diagnosis of tinnitus
 
Good question!

If these overactive fusiform cells is what is putting our hearing into overdrive due to the lack of cochlear input and makes us hear more than there is sound in the environment, then I would say yes, signal timing does affect hearing. This is an obvious conclusion you can make if you subscribe to this theory.

I do believe that somato-auditory stimulation will help alleviate tinnitus. But I worry that one may have to use it all the time unless the cochlear input is restored. So at least in people with hearing loss, I think that restoring cochlear input by repairing the hair cells and neural cells should be the first step. If you achieve good results with that, you may not even need the somato-auditory stimulation at all. Your tinnitus may fade away all on its own.

So we now have two groups of scientists that are trying to address the problem from two different angles. One is trying to restore auditory input at the cochlea, and another is going in opposite direction and trying to calm down overactive neurons. Perhaps the solution is somewhere in-between the two or double folded? This makes me feel optimistic about the future.

The only thing missing that I would like to see more development on is:
1. Objective diagnosis of hearing loss
2. Objective diagnosis of tinnitus

Thank you for answering, I figured Susan Shores device would have to make a compensation for the past few months due to tinnitus and abnormal loudness hyperacusis essentially being the brain acting as an internal hearing aid.

However unlike most people here I'd rather keep my current volume tinnitus then worse hearing.
 
I figured Susan Shores device would have to make a compensation for the past few months due to tinnitus and abnormal loudness hyperacusis essentially being the brain acting as an internal hearing aid.
I am not 100% sure what you are saying here. I can see how hyperacusis is this abnormal perception of loudness, and how tinnitus and hyperacusis might be the result of the brain acting as an internal hearing aid. But what kind of compensation are you talking about, in relation to "past few months"?
 
I am not 100% sure what you are saying here. I can see how hyperacusis is this abnormal perception of loudness, and how tinnitus and hyperacusis might be the result of the brain acting as an internal hearing aid. But what kind of compensation are you talking about, in relation to "past few months"?

There are two kinds of hyperacusis that have separate properties enough to where they deserve their own name.

1: Abnormal amplification hyperacusis :
Tinnitus and abnormal amplification hyperacusis start out are two sides of the same coin. The brain is listening harder to damaged hair cell regions like an internal hearing aid and this causes two problems

Spontaneous neuronal firing in the auditory brain (tinnitus) and amplification of non damaged frequencies by accident making them abnormally loud. (abnormal amplification hyperacusis)

2: Painful hyperacusis/Noxacusis
= Hyperacusis with pain caused by damage to the outer hair cells and a small a mount of nerve fibres in the OHC act like pain receptors in response to insult.
https://neurosciencenews.com/inner-ear-neuron-damage-3033/



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Also how will painful hyperacusis sufferers be able to get treatment for UoM's device?
 
There are two kinds of hyperacusis that have separate properties enough to where they deserve their own name.

1: Abnormal amplification hyperacusis :
Tinnitus and abnormal amplification hyperacusis start out are two sides of the same coin. The brain is listening harder to damaged hair cell regions like an internal hearing aid and this causes two problems

Spontaneous neuronal firing in the auditory brain (tinnitus) and amplification of non damaged frequencies by accident making them abnormally loud. (abnormal amplification hyperacusis)

2: Painful hyperacusis/Noxacusis
= Hyperacusis with pain caused by damage to the outer hair cells and a small a mount of nerve fibres in the OHC act like pain receptors in response to insult.
https://neurosciencenews.com/inner-ear-neuron-damage-3033/

View attachment 19002

Also how will painful hyperacusis sufferers be able to get treatment for UoM's device?
Is it just me or does that nerve cell body look like an eye? o_O :)

Informative article! And nice infographic! But I could not help noticing the last paragraph.

The good news, Fuchs says, is that the analogies with pain elsewhere in the body provide guidance for future studies. For example, a compound that suppresses pain-sensing nerve cells elsewhere, also prevented type II afferent neurons from firing in response to outer hair cell death. At present, Fuchs cautions, this is a restricted experimental result. But, it provides a "proof of concept" for treating pain associated with inner ear damage. And the Fuchs laboratory plans to explore this question in their ongoing research.

Also how will painful hyperacusis sufferers be able to get treatment for UoM's device?
I have no good answer to that. But I totally understand your concern. I would imagine that they need to repair any damage they may have, before they are able to calm down their nervous system.
 
If you love listening to podcasts, tune into the ATA's latest episode on groundbreaking research using bimodal stimulation to treat a specific type of tinnitus.

Episode 7 features Dr. Susan Shore, a professor of neuroscience at the University of Michigan, who has devoted her career to studying the mechanisms underlying tinnitus generation.

In the 25-minute podcast, Dr. Shore discusses the decades-long research and the painstaking details that produced a viable technique for decreasing perception of tinnitus loudness and its accompanying distress. While the subject is highly technical, it reveals both the complexity of validating findings and the promise of future discoveries. Shore also touches on the work of other labs, as well as her other research on synaptopathy, or hidden hearing loss.


To listen to the podcast, click here or visit our website at www.ata.org.
 
If you love listening to podcasts, tune into the ATA's latest episode on groundbreaking research using bimodal stimulation to treat a specific type of tinnitus.

Episode 7 features Dr. Susan Shore, a professor of neuroscience at the University of Michigan, who has devoted her career to studying the mechanisms underlying tinnitus generation.

In the 25-minute podcast, Dr. Shore discusses the decades-long research and the painstaking details that produced a viable technique for decreasing perception of tinnitus loudness and its accompanying distress. While the subject is highly technical, it reveals both the complexity of validating findings and the promise of future discoveries. Shore also touches on the work of other labs, as well as her other research on synaptopathy, or hidden hearing loss.

To listen to the podcast, click here or visit our website at www.ata.org.
This looks awesome I'm tuning in as soon as I can.
 
Does anyone feel this will be helpful to those who suffer from Virus induced tinnitus? Hearing loss due to the shingles virus. I don't see why not if all the same parts are affected. Great thread by the way, the first real hope that seems tangible.
 

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