- Aug 5, 2019
- 1,852
- Tinnitus Since
- 05/2019
- Cause of Tinnitus
- Autoimmune hyperacusis from Sjogren's Syndrome
I realize the title is pretty simple, but let me elaborate. I often hear people talk about the following ideas:
1) Ototoxicity
2) Temporary spike from medicine
3) Temporary spike from stress, high blood pressure, exercise, etc.
I understand ototoxicity -- the drug is damaging the cochlea, making tinnitus worse. But I don't understand why subjective tinnitus increases as a result of medication side effects and increased blood pressure. Tinnitus is, by definition, the brain making up a signal. Why does it make up more of the signal because of pressure? I'm not talking about pulsatile tinnitus, but just tinnitus in general. Does the increased pressure on the inner ear cause it to signal differently? Does the brain react differently to increased cranial pressure?
What motivated me to ask this is I keep reading articles saying that high blood pressure makes one "notice tinnitus more." This infers that there is no damage being done. But then why is our brain perceiving it more?
I just started taking flonase and my tinnitus spiked. Assuming this isn't ototoxicity, why does this occur? I ask these questions because I have a severe case of hyperacusis without much medical direction. I want to gain more of an understanding of my problems. Thanks.
1) Ototoxicity
2) Temporary spike from medicine
3) Temporary spike from stress, high blood pressure, exercise, etc.
I understand ototoxicity -- the drug is damaging the cochlea, making tinnitus worse. But I don't understand why subjective tinnitus increases as a result of medication side effects and increased blood pressure. Tinnitus is, by definition, the brain making up a signal. Why does it make up more of the signal because of pressure? I'm not talking about pulsatile tinnitus, but just tinnitus in general. Does the increased pressure on the inner ear cause it to signal differently? Does the brain react differently to increased cranial pressure?
What motivated me to ask this is I keep reading articles saying that high blood pressure makes one "notice tinnitus more." This infers that there is no damage being done. But then why is our brain perceiving it more?
I just started taking flonase and my tinnitus spiked. Assuming this isn't ototoxicity, why does this occur? I ask these questions because I have a severe case of hyperacusis without much medical direction. I want to gain more of an understanding of my problems. Thanks.