my 2 cents- i have years of experience with Cymbalta. In fact, it helped me the first time so i haven't tried anything else. I tapered it off also quite easily, after 2,5 years of 30mg of use. Can't quit cold turkey though.
At the time i did not develope H, after i quit C and had some acoustic traumas i did not recover as i normally would. My theory is that C protected me. SNRI or serotonine reuptake inhibitor is widely known for pain easer as well. I'm on C again right now and my H has not come back after LLLT, about a year ago. Ofc i'm being overly paranoid, protective and careful these days. I would get on, at least somekind of a SNRI/SSRI-asap!
"If I recall correctly, 95% of Seratonin is produced in the gut"- then i would eat bananas all day, take sun and eat L-Tryptophan. But it has not been major help as i have needed the SNRI/SSRI to sensitize 5-HT receptor.
http://www.dizziness-and-balance.com/disorders/hearing/hyperacusis.htm
"Hyperacusis is very often associated with migraine (where it is called phonophobia). Because migraine is extremely common (10% of the population), it likely accounts for at least 90% of all hyperacusis. Migraine involves serotonin pathways, which are also postulated to be at the root of central hyperacusis"
food for thought
At the time i did not develope H, after i quit C and had some acoustic traumas i did not recover as i normally would. My theory is that C protected me. SNRI or serotonine reuptake inhibitor is widely known for pain easer as well. I'm on C again right now and my H has not come back after LLLT, about a year ago. Ofc i'm being overly paranoid, protective and careful these days. I would get on, at least somekind of a SNRI/SSRI-asap!
"If I recall correctly, 95% of Seratonin is produced in the gut"- then i would eat bananas all day, take sun and eat L-Tryptophan. But it has not been major help as i have needed the SNRI/SSRI to sensitize 5-HT receptor.
http://www.dizziness-and-balance.com/disorders/hearing/hyperacusis.htm
"Hyperacusis is very often associated with migraine (where it is called phonophobia). Because migraine is extremely common (10% of the population), it likely accounts for at least 90% of all hyperacusis. Migraine involves serotonin pathways, which are also postulated to be at the root of central hyperacusis"
food for thought