In this article a figure of 54.7 percent was mentioned. Amazing.
Quote: "Sanchez said they found the prevalence of tinnitus was higher in teens than in the general population: 54.7 percent (identified through a questionnaire)"
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I heard about the odd story of people's Tinnitus being resolved after a new noise exposure, maybe the disconnecting MOC-efferents is what happened in that case. Seeing all the possible combinations/outcomes, I wouldn't recommend trying to replicate that, because it seems like playing russian roulette with the synapses. It might however be a (rare) possibility if this is true.
Two questions:
1) What is the difference between hidden hearing loss and sensorineural hearing loss?
2) Is hidden hearing loss strictly noise-induced?
Two questions:
1) What is the difference between hidden hearing loss and sensorineural hearing loss?
2) Is hidden hearing loss strictly noise-induced?
Maybe the type-1 afferent is able to reconnect, but the MOC-efferent grows into place at a faster pace by nature. Maybe it depends on the severity of the noise trauma and are the type-1 efferents "swung away" from the IHC further than a nearby MOC-efferent if the trauma is severe enough? Maybe some chemical is involved in determining or suppressing which type of synapse is allowed to connect (and maybe this chemical balance is disturbed)?
Maybe the type-1 afferents reconnect to the IHC at a later time, resulting in Tinnitus-reduction due to the already connected MOC-efferents (explaining the case "Tinnitus reduction after a while but not really resolved (got more into the background)" like often described)?
Lots of "maybe's", I know, but interesting stuff to think about. A lot of different combinations in connections are possible and it seems these connections and the status of each individual IHC changes a lot throughout life (similar to the brain-synapse connection analogy), making Tinnitus in its current form and intensity not necessarily permanent in that way, but ever changing. I wonder if MOC-efferents are able to disconnect or be disconnected in any way after connecting to the IHC...
I heard about the odd story of people's Tinnitus being resolved after a new noise exposure, maybe the disconnecting MOC-efferents is what happened in that case. Seeing all the possible combinations/outcomes, I wouldn't recommend trying to replicate that, because it seems like playing russian roulette with the synapses. It might however be a (rare) possibility if this is true.
A conductance test has been performed.Maybe you don't have a problem of inner ear but a problem of middle ear
Yes. The typical noise induced type. Notch at the important frequencies for understanding speech.You have hearing loss ?
Sometimes I feel medical science is still so far away from understanding the workings of the inner ear
Stem cells have the information to repair in the right order. That is why I think there could be something interesting happening there. I know this is a very simple way of looking at it. But still?it's just a matter of repairing damage in the right order and finding a way to not cause any further damage.
When slight hearing loss is able to improve, the principle could be the same for more severe hearing loss and associated discomforts. (Discomforts is putting it mildly )-.I honestly think it will be within 5 years before there is treatment available, including for those with slight hearing loss.
That's just it. Hidden hearing loss does not show on an audiogram. )-:I have no non hidden hearing-loss, audiograms are perfect
Yes. The typical noise induced type. Notch at the important frequencies for understanding speech.
The MOCs may resume their earlier position on the inner hair cell but from everything we have seen, there is no functional synapse. In other words, the chemical factor that was keeping them away from the inner hair cell is gone but that doesn't mean the MOCs have the means to talk to the inner hair cell. Also, keep in mind that the net effect of MOC activation is to dampen electrical currents in hair cells (at least in outer hair cells).
Hello @Vinnitus .
Yes a school example of NIHL. 4Khz. Also "wear and tear". So above 8 kHz sloping down. The latter is age related, but still the same principle. Loss of haircells/synapses.
After the incident I did have fullness in the (mostly) right ear. This ear is most effected.
I do still have this feeling. Although it is abating very slowly.
The feeling of fullness was continuous and is slowly starting to get intermittent. That's is an improvement I notice.
I often have the feeling of tinnitus in my ears and do not hear a loud tinnitus. As if my tinnitus frequency is too high to detect. I firmly believe that is what is happening.
More like a year.After what time period did your aural fullness start to become intermittent? weeks, months?
9.5 kHz. Occasionally (seldom) 4-5 kHz.Tinnitus tone you would classify as 4kHz? Or do you only have higher tones (>8kHz)?
Yes. So do I.but I wonder whether they will be able to repair damaged auditory nerve fibres before hair cell regeneration.
Yes. So do I.
Also I would not be surprised that slowly science is starting to find that our ears are much, much more prone to damage (hidden hearing loss) than previous assumed. An effect of this realization is that maximum safe sound levels will go down.
Our ears are not robust at all. Not so surprising if you look at the delicate structure of the inner ear.
It is amazing that you experience distortion, tinnitus and hyperacusis with a perfect audiogram.
What does this tell you about the value of an audiogram?
I think if you are this young, it is not unrealistic to hope for future cures.
Don't under estimate anxiety.it's just your anxiety'.
Don't under estimate anxiety.
I personally experience(d) that anxiety can do a lot of "damage". Hopefully reversible.