Hidden Hearing Loss, Tinnitus and Trouble Hearing Conversations in Noise

In this article a figure of 54.7 percent was mentioned. Amazing.
Quote: "Sanchez said they found the prevalence of tinnitus was higher in teens than in the general population: 54.7 percent (identified through a questionnaire)"
End quote.
I did a search on the internet Epidemic and hearing loss. These two seem to go well together. Plenty articles.
Perhaps one good thing coming from this is that it is more difficult to ignore.
It is costing money! Money is what makes our world go round.
 
In this article a figure of 54.7 percent was mentioned. Amazing.
Quote: "Sanchez said they found the prevalence of tinnitus was higher in teens than in the general population: 54.7 percent (identified through a questionnaire)"
.

I think (hope) this 54.7% stands for kids with T incidents and not the chronic teenage T sufferers. If that was for chronic then it's a disaster.
 
I heard about the odd story of people's Tinnitus being resolved after a new noise exposure, maybe the disconnecting MOC-efferents is what happened in that case. Seeing all the possible combinations/outcomes, I wouldn't recommend trying to replicate that, because it seems like playing russian roulette with the synapses. It might however be a (rare) possibility if this is true.

Suffering from mild hearing loss and tinnitus after prolonged noises exposures in the past, I seem since a few months ago to have actually decrease of the tinnitus and the mild hyperacusis (along with loss of hearing dynamics a sparkle) after a possible new ear trauma because it coincides with a rifle shooting practice from an indoor launch pad with large caliber (very loud and short noises which makes lots of vibrations) though I had very higher range ear protections. But if I could step back in time I would keep higher tinnitus and better hearing because now I feel no more tiny noise exposure risk can be taken. If only all those knowledges or even hypothesis about inner ear neural zones fragility had been spread the last decades. Even today Ents and general practitioners are relunctant to open their minds to this reality slowly beeing recognized.
 
Two questions:

1) What is the difference between hidden hearing loss and sensorineural hearing loss?
2) Is hidden hearing loss strictly noise-induced?
 
Two questions:

1) What is the difference between hidden hearing loss and sensorineural hearing loss?
2) Is hidden hearing loss strictly noise-induced?

Hidden hearing loss is just what is not detected on a standard audiogram. Hidden hearing loss is sensorineural hearing loss.

Is it strictly noise-induced? Probably not.
 
I think I'm genetically screwed when it comes to earth health and tolerance. I know at 13 I wen't to these little teen clubs and music was loud. Had ringing numerous times that wen't away. After that phase not much noise issue until 18 where I wen't to electronic festival and got a temp. threshold shift and noticeable T. Bad ears, damage from 13 contributed, who knows.

I got a hearing test and was fine. Perfect actually. So, mine clearly is above testable ranges. Problem for me, is mine is getting worse. After 18 getting my first perm. T, I was fine until age 26 where a loud car gave my hyperacusis and a wood saw outside my window woke me up, I got hit for solid 10 seconds. 5 days later I wen't to bed notice T super loud, never was same. Last year or so, T changing, bad hyperacusis and very little progress as motorycles, jets and other noises screw over me ears. I was semi-stable for awhile recently then i got hit by 114db for a whopping 1 second. Worse T almost 2 weeks in...how do you even explain that from a brief exposure?

All with perfect 6 year old hearing. So, I must be extremely damaged in non-testing ranges. If a brief 114 in my ear can change my T and world...how can I continue forward. This noise was from knocking something over, my own home...safe supposedly.

I'm screwed and I think the hidden hearing loss I got at young age screwed me over with my ears able to handle noise and repair as an adult. I admire everyone with T that just stays consistent and even little exposures don't change it.
 
Two questions:

1) What is the difference between hidden hearing loss and sensorineural hearing loss?
2) Is hidden hearing loss strictly noise-induced?

Hidden hearing loss generally refers to a hearing deficit that goes undetected (i.e. "hides") behind an otherwise normal report from the audiologist. Increasingly, it is being used to refer to a difficulty tracking conversations and other complex sounds in high levels of background noise or in rooms with a lot of reverb.

Sensorineural hearing loss describes a possible biologic origin of the hearing loss. Some hearing loss is metabolic (in which the ear's "battery" is not functioning properly), some is conductive (in which sound is not transmitted normally through the outer ear and middle ear) and some is sensorineural (which refers to a dysfunction of sensory hair cells in the cochlea or to the neurons that convert these signals into electrical activity that creates the perception of sound).

As for your second question, no, hidden hearing loss need not be strictly noise-induced. It could be a consequence of normal aging, ototoxic drugs etc. The basic idea is that if you damage the neural connections without damaging the sensory hair cells, problems will pop up in listening conditions that are not tested in a routine visit to the audiologist. With that said, noise exposure is probably the most direct route to hidden hearing loss (and tinnitus).
 
Maybe the type-1 afferent is able to reconnect, but the MOC-efferent grows into place at a faster pace by nature. Maybe it depends on the severity of the noise trauma and are the type-1 efferents "swung away" from the IHC further than a nearby MOC-efferent if the trauma is severe enough? Maybe some chemical is involved in determining or suppressing which type of synapse is allowed to connect (and maybe this chemical balance is disturbed)?

Maybe the type-1 afferents reconnect to the IHC at a later time, resulting in Tinnitus-reduction due to the already connected MOC-efferents (explaining the case "Tinnitus reduction after a while but not really resolved (got more into the background)" like often described)?

Lots of "maybe's", I know, but interesting stuff to think about. A lot of different combinations in connections are possible and it seems these connections and the status of each individual IHC changes a lot throughout life (similar to the brain-synapse connection analogy), making Tinnitus in its current form and intensity not necessarily permanent in that way, but ever changing. I wonder if MOC-efferents are able to disconnect or be disconnected in any way after connecting to the IHC...

I heard about the odd story of people's Tinnitus being resolved after a new noise exposure, maybe the disconnecting MOC-efferents is what happened in that case. Seeing all the possible combinations/outcomes, I wouldn't recommend trying to replicate that, because it seems like playing russian roulette with the synapses. It might however be a (rare) possibility if this is true.

The MOCs may resume their earlier position on the inner hair cell but from everything we have seen, there is no functional synapse. In other words, the chemical factor that was keeping them away from the inner hair cell is gone but that doesn't mean the MOCs have the means to talk to the inner hair cell. Also, keep in mind that the net effect of MOC activation is to dampen electrical currents in hair cells (at least in outer hair cells).
 
Sometimes I feel medical science is still so far away from understanding the workings of the inner ear :(

I honestly think it will be within 5 years before there is treatment available, including for those with slight hearing loss. Hearing loss has been cured in mice in multiple different ways, it's just a matter of repairing damage in the right order and finding a way to not cause any further damage. The latter, I think, will be the easiest because it would involve microscopic technology, and 5 years is enough for required innovation.
 
it's just a matter of repairing damage in the right order and finding a way to not cause any further damage.
Stem cells have the information to repair in the right order. That is why I think there could be something interesting happening there. I know this is a very simple way of looking at it. But still?
I honestly think it will be within 5 years before there is treatment available, including for those with slight hearing loss.
When slight hearing loss is able to improve, the principle could be the same for more severe hearing loss and associated discomforts. (Discomforts is putting it mildly )-;).
Sometimes I agree and think after reading about the "breakthroughs" I see indications a treatment could be not too far away.
But sometimes I read what is not yet known about, what sounds like basic processes in the inner ear, I loose this confidence.
I have no non hidden hearing-loss, audiograms are perfect
That's just it. Hidden hearing loss does not show on an audiogram. )-:
 
Yes. The typical noise induced type. Notch at the important frequencies for understanding speech.

Hello @Reinier

Out of curiosity; do you have the classical "noise-notch" at 4Khz or at another frequency? Also, did you experience aural fullness directly after the incident and do you still experience it? If so, has the fullness been intermittent or continuous in nature?
 
The MOCs may resume their earlier position on the inner hair cell but from everything we have seen, there is no functional synapse. In other words, the chemical factor that was keeping them away from the inner hair cell is gone but that doesn't mean the MOCs have the means to talk to the inner hair cell. Also, keep in mind that the net effect of MOC activation is to dampen electrical currents in hair cells (at least in outer hair cells).

Thank you for this information. I suppose you mean the MOC-efferent moves into place on the former position of the type-1 afferent in this case? If the MOC-efferents are not able to talk to the IHC, it would invalidate my earlier theory regarding reconnecting MOC-efferents. Maybe the loss of type-1 afferents is enough though...

I have been reading into discordant dysfunction theory for Tinnitus, which proposes imbalance of signalling coming from the IHCs and the OHCs for a certain frequency resulting in the perception of Tinnitus in the dorsal cochlear nucleus (DCN). Is this theory (still) considered viable? If so, could the loss of type-1 afferent connections to the IHC in hidden hearing loss perhaps cause this same imbalance in de DCN and as such be a possible etiology for Tinnitus?
 
Hello @Vinnitus .
Yes a school example of NIHL. 4Khz. Also "wear and tear". So above 8 kHz sloping down. The latter is age related, but still the same principle. Loss of haircells/synapses.
After the incident I did have fullness in the (mostly) right ear. This ear is most effected.
I do still have this feeling. Although it is abating very slowly.
The feeling of fullness was continuous and is slowly starting to get intermittent. That's is an improvement I notice.
I often have the feeling of tinnitus in my ears and do not hear a loud tinnitus. As if my tinnitus frequency is too high to detect. I firmly believe that is what is happening.
 
Hello @Vinnitus .
Yes a school example of NIHL. 4Khz. Also "wear and tear". So above 8 kHz sloping down. The latter is age related, but still the same principle. Loss of haircells/synapses.
After the incident I did have fullness in the (mostly) right ear. This ear is most effected.
I do still have this feeling. Although it is abating very slowly.
The feeling of fullness was continuous and is slowly starting to get intermittent. That's is an improvement I notice.
I often have the feeling of tinnitus in my ears and do not hear a loud tinnitus. As if my tinnitus frequency is too high to detect. I firmly believe that is what is happening.

Interesting. After what time period did your aural fullness start to become intermittent? weeks, months? Also, do you experience a Tinnitus tone you would classify as 4kHz? Or do you only have higher tones (>8kHz)? I also experience a weird sensation in the ear indicating the presence of Tinnitus or the abnormality causing it, even if the Tinnitus is indeed very low.
 
After what time period did your aural fullness start to become intermittent? weeks, months?
More like a year.
Tinnitus tone you would classify as 4kHz? Or do you only have higher tones (>8kHz)?
9.5 kHz. Occasionally (seldom) 4-5 kHz.
I realize tinnitus actually means there has to be sound. But there is so much more to damaged inner ears.
Sometimes I get the feeling that a very sharp scalpel is cutting through my brains.
I hate this condition:mad:
 
journals.lww.com/thehearingjournal/Fulltext/2016/11000/Hidden_Hearing_Loss___A_Clinician_s_Perspective.2.aspx

Again: this suggests it needs administration close after noise exposure:
"successful promotion of auditory neuronal survival via neurotrophin delivery, gene therapy, and electric stimulation, among others, has been thoroughly demonstrated in animal models."

But I am glad with the research they are doing. Who knows what they will learn from this approach.
 
This is a very base speculation, but I wonder whether they will be able to repair damaged auditory nerve fibres before hair cell regeneration. I am still waiting on an ENT appointment but I am pretty sure I suffer from this "hidden hearing loss" - have had tinnitus for the past five years although I noticed no other problems with my hearing, in fact my hearing still felt pretty pristine, however, recently it's been awful. My audiogram from last week came up as perfect but in the past six months or so I have had hyperacusis, irritated ears, distortion, and most disconcertingly the persistent sense of derealisation. Everything sounds dulled and 'far away'. I'm only 20 years old too so feel like the best part of my life is over.
 
but I wonder whether they will be able to repair damaged auditory nerve fibres before hair cell regeneration.
Yes. So do I.

Also I would not be surprised that slowly science is starting to find that our ears are much, much more prone to damage (hidden hearing loss) than previous assumed. An effect of this realization is that maximum safe sound levels will go down.
Our ears are not robust at all. Not so surprising if you look at the delicate structure of the inner ear.

It is amazing that you experience distortion, tinnitus and hyperacusis with a perfect audiogram.
What does this tell you about the value of an audiogram?

I think if you are this young, it is not unrealistic to hope for future cures.
 
Yes. So do I.

Also I would not be surprised that slowly science is starting to find that our ears are much, much more prone to damage (hidden hearing loss) than previous assumed. An effect of this realization is that maximum safe sound levels will go down.
Our ears are not robust at all. Not so surprising if you look at the delicate structure of the inner ear.

It is amazing that you experience distortion, tinnitus and hyperacusis with a perfect audiogram.
What does this tell you about the value of an audiogram?

I think if you are this young, it is not unrealistic to hope for future cures.

Yeah, ears are so delicate. And not many people realise that. Even a family friend with 30 years experience as a teacher of deaf kids dismissed me when I said I definitely had damage telling me 'your hearing is perfect as you know from your audiogram - it's just your anxiety'.
 

Log in or register to get the full forum benefits!

Register

Register on Tinnitus Talk for free!

Register Now