My Posting Place

true you make a point. I have heard some people hear say their parents have tinnitus and never gave a damn about it.
That's true. One of my friends' dad has tinnitus but said he didn't really care. I'm personally a little autistic and naturally anxious. I think that maybe you have to be mentally a bit "sick" to care about tinnitus or really have it bother you (talking about mild tinnitus here). Just a thought.
 
That's true. One of my friends' dad has tinnitus but said he didn't really care. I'm personally a little autistic and naturally anxious. I think that maybe you have to be mentally a bit "sick" to care about tinnitus or really have it bother you (talking about mild tinnitus here). Just a thought.
noise induced pain even in very mild forms is a devastating torture, because your entire life is going to be an on and off struggle while pain interferes with your rational descion making, social interaction, fun activities and relaxation.

Don't listen to the people who "manage hyperacusis" they are just tricking themselves.
 
Tonal audiogram history.

The concept of a frequency versus sensitivity (amplitude) audiogram plot of human hearing sensitivity was conceived by German physicist Max Wien in 1903. The first vacuum tube implementations, November 1919, two groups of researchers — K.L. Schaefer and G. Gruschke, B. Griessmann and H. Schwarzkopf — demonstrated before the Berlin Oto-logical Society two instruments designed to test hearing acuity. Both were built with vacuum tubes. Their designs were characteristic of the two basic types of electronic circuits used in most electronic audio devices for the next two decades. Neither of the two devices was developed commercially for some time, although the second was to be manufactured under the name "Otaudion." The Western Electric 1A, developed by <who> was built in 1922 in the United States. It was not until 1922 that otolaryngologist Dr. Edmund P. Fowler, and physicists Dr. Harvey Fletcher and Robert Wegel of Western Electric Co. first employed frequency at octave intervals plotted along the abscissa and intensity downward along the ordinate as a degree of hearing loss. Fletcher et al. also coined the term "audiogram" at that time.

With further technologic advances, bone conduction testing capabilities became a standard component of all Western Electric audiometers by 1928.
 
FUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUUCK
@JohnAdams agree??
 
https://www.sciencedaily.com/releases/2015/11/151109182050.htm

ITT we debate if regenerating OHC's or repairing their ribbon synapses will stop this peripheral form of "noise induced pain"

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Outer hair cells are the canaries in the coal mine for the inner ear, in that they're the first cells to die due to loud noise, age or other factors," says Fuchs. "Since they can't regenerate, their death leads to permanent hearing loss." So one possible role for type II afferents, he adds, would be to warn the brain of impending damage to outer hair cells.

It was known that nearby supporting cells respond to outer hair cell damage by increasing their inner calcium levels and releasing the chemical messenger ATP. Fuchs' team knew that type II afferent neurons can respond to ATP, so they damaged outer hair cells while monitoring type II neurons in surgically removed inner ear tissue. Indeed, outer hair cell rupture caused robust excitation of type II neurons.

Fuchs says that the ATP released by the supporting cells is probably what gets the neurons to fire, and the supporting cells might release ATP in response to ATP that leaks out of the ruptured outer hair cells. But he noted that "outer hair cells don't have to rupture to release ATP. Progressive damage caused by loud noises or other stress is enough to increase ATP levels in the fluid of the inner ear.
 
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How does one lower ATP levels in the cochlea, it's needed to here but in noise induced pain it's hypothesized to be excessive, that may explain amplification of noise?

But i'm just hypothesizing please don't view my post as serious facts about how to treat this condition.
 
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Do you even cochlear synaptopathy bro?
 
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WRONG, do you even hyperactivity in dorsal cochlear nucleus after less cochlear input bruh?
 
yes especially the older generation, they just probably think all music sounds like garbage nowadays and that everyone has trouble hearing in background noise.

Slowly losing hearing goes unnoticed because they are conditioned.

This was true for me. The responsibilities I had didn't care if I had tinnitus or not. "I didn't have time to be sick." If I could still fulfill my responsibilities it didn't matter if my hearing was 50%.

Until another noise induced incident caused my T to triple. This was unbearable. I gave up all my hobbies except for work related responsibilities. I tried as hard as I could to work at the same capacity. After several years I cracked, fell flat on my face and lost everything.
 
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..................
it's still complexing this person does not have a history of noise exposure.

@Charles_Liberman
 
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I'm literally using MPP as an information dump and a place to feel sorry for myself and basterdize generations before us that didn't advocate for research.
 
This was unbearable. I gave up all my hobbies except for work related responsibilities.
Sh!t. I'm really sorry man. I'm in the same position. I basically gave up everything besides work , which I'm holding on to out of bare necessity.
I tried as hard as I could to work at the same capacity. After several years I cracked, fell flat on my face and lost everything.
I did reduce my work hours by about 25-30%. I hope I won't completely go crazy but I might. This sh!t is insufferable. Can we get a mutual hug.
 
Sh!t. I'm really sorry man. I'm in the same position. I basically gave up everything besides work , which I'm holding on to out of bare necessity.

I did reduce my work hours by about 25-30%. I hope I won't completely go crazy but I might. This sh!t is insufferable. Can we get a mutual hug.
You got it *Hug* it's pretty much all I do now too. What a life @ 28 years, should be my prime.
 
View attachment 25882

I'm literally using MPP as an information dump and a place to feel sorry for myself and basterdize generations before us that didn't advocate for research.
I'm pretty sure these are very valid.
Research? Unless you were missing an arm or leg, had a sucking chest wound or covered in blood, no one gave a crap if you were sick or injured.
Ugggh. What period are you referring to, circa?
 
Sh!t. I'm really sorry man. I'm in the same position. I basically gave up everything besides work , which I'm holding on to out of bare necessity.

I did reduce my work hours by about 25-30%. I hope I won't completely go crazy but I might. This sh!t is insufferable. Can we get a mutual hug.
You got it *Hug* it's pretty much all I do now too. What a life @ 28 years, should be my prime.
No one understands. It's an invisible and undetectable affliction. No one understands our suffering except those of us here with it.

That's why I'm here. You guys make me feel "normal".
 
No one understands. It's an invisible and undetectable affliction. No one understands our suffering except those of us here with it.

That's why I'm here. You guys make me feel "normal".
Exactly, my body is otherwise fine but I feel ill pretty much everyday, I shake from anxiety when I wake up and sleep is my friend.
 
Imagine we didn't have TT and only had the f@ggot Jasterdoff Temple © Facebook groups.
Sorry to be harsh but that's exactly what they are.
 
Imagine we didn't have TT and only had the f@ggot Jasterdoff Temple © Facebook groups.
Sorry to be harsh but that's exactly what they are.
We need to join the facebook groups and preach the gospel.

We already won the culture war on the forum.
 
Exactly, my body is otherwise fine but I feel ill pretty much everyday, I shake from anxiety when I wake up and sleep is my friend.
We were all just people living our lives as we were able when we were struck by this BS that is beyond description.
What exactly happened? How did we just end up these miserably unlucky ones to get stuck with this torture? Obviously there's a physical answer (noise trauma etc) but it just boggles my mind how this could happen to me/us, IDK.
 
So why hasn't any lab in America loaded up a syringe of PRP and did a human or animal study on it's affect on hearing loss?

There was one about olfactory regeneration, weirdly enough in an ATA sponsored tinnitus journal and it was successful.

And those olfactory cells are LGG5+ cells too, like what's in our cochleas.
 

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