Anyways, I've been trying to understand this cannabis, and reading about DCN in the animal research, and so forth, and seeing what people say on this forum, and compare with my own experience.
I also check my pharmacist about this, because he is quite brilliant.
So, I have at least 2 things to say.
#1, cannabis is not a mainstay, but if I wake up at 2 a.m. and need to get back to sleep in a hurry, 0.2 g, which is quite a small amount, taken through the vaporizer (set at 190C), acts very instantly, and that kind of dose, for me, does not cause tinnitus to increase. if it does increase, I'm not worried, because cannabis is not known to be ototoxic. I also, have been putting the herb out in the sun for a couple days, since the U.V. will convert much of the thc to cbn (not cbd), and cbn tends to prefer cb2 receptors, so does not interact so much in the DCN. So, that's what I do ymmv.
#2, my pharmacist tells me, that regular taking of cannabis, causes downregulation of the receptors (meaning the body eliminates the receptors, so you have much less receptor, with regular use). so, if you stop taking cannabis, the opposing neurotransmitters, do not have any cannabis to contend with, with actually makes tinnitus get less after stopping for a few days.
#3, based on the enormous variation in cannabinoids, I believe there is a possibility that either 1. a cannabis strain has the optimum balance of various cannabinoids, or 2. harvesting the flowers at the hyper-ripe phase where thc has naturally transformed to cbn, may provide some relief. so I am planning to see what different strains are available, and see if there is a difference due to strains. if this is true, it could be a valuable observation for clinical trial.
personally, myself, I find that if I get a very good sleep, then I wake up both refreshed and ready to tackle the day, and my tinnitus is usually a bit less if I get a good sleep. if I sleep poorly, I can't function, and my tinnitus/hyperacussis is generally much worse, which puts me into a depressed state.