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- Thread starter threefirefour
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Thank you John Adams, for introducing me to Purple Boys, Erlend and Steinjo. Keeping my insanity in good company.
That's bullshit. If it works then you should have the right to buy it online. But yeah, you'll be flying for sure.
nah mute buttons really meant to reduce tinnitus, that was just an ignorant normie comment.
- May 29, 2018
- 1,161
- Tinnitus Since
- 10/2017
- Cause of Tinnitus
- Neck/stress
If it doesn't work I'm dead and i'll just have to make my death as natural as possible. I cannot keep on going like this being mentally gang raped by screeching sounds day and night......I'm traumatised to fuck.
I'm sorry you are suffering. "Tinnitus Since: 10/17" isn't a long time. This is good reason to hope things will become more bearable with time. Give it five years. I still suffer but not 24/7. Occasionally there is a good day that makes things worthwhile. Hang in there brother. We all love you here.
- May 29, 2018
- 1,161
- Tinnitus Since
- 10/2017
- Cause of Tinnitus
- Neck/stress
Thanks dude. You've done so well to keep going. You've been at this for a long time. It's like being trapped in a horrendous acid trip....forever.
Thanks, you're doing well too. No one acknowledges our challenge as much as those of us plagued with it. Looking back, had I been successful at my foolish attempts, this would have been a huge mistake. Thankfully the only thing that ended is the expectation of living without suffering.
. I fucking plan to be back at life. My T is a nasty screech to and today fucking BLEW ASS WOW. Music sounded awful and T was just relentless as hell.
I'm not suggesting anything implied in the above quote. It was over four years before I could even visit this forum without anguish.
With no cure, we only have two choices regarding life with tinnitus: "Take it" or "Leave it".
My rational mind says deal with it.
The days I'm crippled by T, I think to myself I'd rather be dead.
But being dead is too easy and forfeits any future possibilities. We don't live forever anyway, so I'm stubborn enough to suffer to the natural end of things. I'm not letting T win.
Tinnitus can kiss my... grits. The enemy which lives between my ears.
When do we find out about fx-322?
I want to know if type II Outer hair cell nerve fibers send pain signals through the Audiotory nerve to the Dorsal Cochlear nucleus which is then sent to the trigerminal nerve. Then pain is experienced. Hyperacusis with pain is both the trigerminal nerve and audiotory nerve sending pain signals. (noise induced ear and face pain)
What about other triggers for hyperacusis with pain, does hyperactivity in the audiotory brain caused by tinnitus have anything to do with it? Is this loudness, or pain hyperacusis or both?
What about loudness sensitivity in some autism cases or Audiotory processing disorders I believe "loudness hyperacusis" is that same thing. Yet loudness hyperacusis also occurs in acoustic trauma sufferers. In Autism and APD is there hyperactivity in the DCN as well?
Outer hair cells are the cochlea's amplifiers, do damaged outer hair cells also cause some noises to sound too loud combined with massive outerhair cell damage activating the type II afferent nerve fibers? But there also have to be a central pathology.
Some people expose themselves to very loud noises and do massive hearing damage and do not have tinnitus and hyperacusis? There must be a neurological gateway mechanism
I really do believe there is a central and peripheral version of hyperacusis. I wonder if tinnitus is the phantom limb sensation then maybe one form of hyperacusis is the phantom pain to compensate for a damaged audiotory nerve.
Because some people experience chronic ear fullness.
I am also convinced there are 2-3 types of hyperacusis
and I do acknowledge subtypes of tinnitus depending on what part of the brain tinnitus is established in.
We don't exactly whats happening know research is not there yet
I look stupid speculating of things way over any qualification I have but this literally keeps me up at night knowing audiologist and ENT's aren't trying to escalate these questions to bio-medical research to help sufferers. They just shill Tinnitus Retraining Therapy without looking into the depths of the problem
TRT'S creator, Pawel Jasterboff is a quack, his research is from several decadesd ago before anyone knew about type II afferent nerve fibers or the role of the trigerminal nerve in hyperacusis.
He never considering looking at the suspected pathologies in hyperacusis and subtypes (pain and loud) and trying to think about how his treatment of TRT would apply to any of them. Once again the research wasn't therea t the time.
He just shilled TRT to the world and obviously predatory audiologist that sell hearing aids for a living would have no problem picking up dogma. Audiology is literally a extremely outdated pseudo-science field
What about other triggers for hyperacusis with pain, does hyperactivity in the audiotory brain caused by tinnitus have anything to do with it? Is this loudness, or pain hyperacusis or both?
What about loudness sensitivity in some autism cases or Audiotory processing disorders I believe "loudness hyperacusis" is that same thing. Yet loudness hyperacusis also occurs in acoustic trauma sufferers. In Autism and APD is there hyperactivity in the DCN as well?
Outer hair cells are the cochlea's amplifiers, do damaged outer hair cells also cause some noises to sound too loud combined with massive outerhair cell damage activating the type II afferent nerve fibers? But there also have to be a central pathology.
Some people expose themselves to very loud noises and do massive hearing damage and do not have tinnitus and hyperacusis? There must be a neurological gateway mechanism
I really do believe there is a central and peripheral version of hyperacusis. I wonder if tinnitus is the phantom limb sensation then maybe one form of hyperacusis is the phantom pain to compensate for a damaged audiotory nerve.
Because some people experience chronic ear fullness.
I am also convinced there are 2-3 types of hyperacusis
and I do acknowledge subtypes of tinnitus depending on what part of the brain tinnitus is established in.
We don't exactly whats happening know research is not there yet
I look stupid speculating of things way over any qualification I have but this literally keeps me up at night knowing audiologist and ENT's aren't trying to escalate these questions to bio-medical research to help sufferers. They just shill Tinnitus Retraining Therapy without looking into the depths of the problem
TRT'S creator, Pawel Jasterboff is a quack, his research is from several decadesd ago before anyone knew about type II afferent nerve fibers or the role of the trigerminal nerve in hyperacusis.
He never considering looking at the suspected pathologies in hyperacusis and subtypes (pain and loud) and trying to think about how his treatment of TRT would apply to any of them. Once again the research wasn't therea t the time.
He just shilled TRT to the world and obviously predatory audiologist that sell hearing aids for a living would have no problem picking up dogma. Audiology is literally a extremely outdated pseudo-science field
Are there people who have hyperactivity in their DCN other suspected brain regions without tinnitus, but hyperacusis('s)? or vice versa?
Is this being investigated? are there people actively trying to figure out the pathologist of hyperacusis and tinnitus, could these questions have already been answered burried away on pubmed or waiting to be published in Prof Liberman or Prof Shorre's next paper. 2018 was a good year for tinnitus and otological research and Jaterboff knows his days are numbered.
I know researchers come browse here on there lunch break every now and then
So I might as well throw in the last question " will regenerating hair cells cause new ribbon synapses to grow on pre-existing damaged hair cells?"
Is this being investigated? are there people actively trying to figure out the pathologist of hyperacusis and tinnitus, could these questions have already been answered burried away on pubmed or waiting to be published in Prof Liberman or Prof Shorre's next paper. 2018 was a good year for tinnitus and otological research and Jaterboff knows his days are numbered.
I know researchers come browse here on there lunch break every now and then
So I might as well throw in the last question " will regenerating hair cells cause new ribbon synapses to grow on pre-existing damaged hair cells?"
Red
Member
And I will leave it at that.
Here's where ill be- post T
An echoic chamber- We wont be strangers
I will take a piss and there will be no HISS! WOO
An echoic chamber- We wont be strangers
I will take a piss and there will be no HISS! WOO
"One of the quietest rooms in the country, also known as the Dead Room." Please don't lock me up in there with T.
I think I'd do it. I hear my T through most shit anyways. I plan on doing a sensory deprivation float soon.
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