1: Would curing sensorineural hearing loss reduce tinnitus if the tinnitus was caused by sensorineural hearing loss?
2: Have they made progress investigating what role GABA has to play in tinnitus patients and if increasing GABA helps?
3: Is there evidence yet of tinnitus having a gateway mechanism, why some individuals with SNHL get tinnitus and others don't?
4: With hearing loss and related problems like tinnitus and painful hyperacusis being common among soldiers, veterans and even the general population how can we get the Governments to show an interest in biomedical research to treat these conditions?
5: Do ENT's do a disservice by ignoring hidden hearing loss and keeping the public ignorant about the condition they suffer from?
6: Is hyperactivity in the DCN or other regions of the brain after hearing loss physical evidence of tinnitus or is this just a speculative hypothesis?
7: How do we get more funding for biomedical research to get sufferers real help, instead of most budget money going towards CBT, TRT type therapies that aren't curative in the long run?
8: Is hyperacusis with pain and hyperacusis with loudness amplification two separate conditions or subtypes of the same condition? I have read hypothesis that suggest this. They seem to have totally different properties. I have heard researchers use words like noxacusis to refer to painful hyperacusis, and recruitment to refer to loudness amplification. Hyperacusis with pain is a horrifying condition and it's mechanisms need to be understood in order of a treatment.
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